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Also known as: 472-61-7, Ovoester, All-trans-astaxanthin, Astareal, 3,3'-dihydroxy-beta,beta-carotene-4,4'-dione, Astaxanthine
Molecular Formula
C40H52O4
Molecular Weight
596.8  g/mol
InChI Key
MQZIGYBFDRPAKN-UWFIBFSHSA-N
FDA UNII
8XPW32PR7I

Astaxanthin
Astaxanthin is a natural and synthetic xanthophyll and nonprovitamin A carotenoid, with potential antioxidant, anti-inflammatory and antineoplastic activities. Upon administration, astaxanthin may act as an antioxidant and reduce oxidative stress, thereby preventing protein and lipid oxidation and DNA damage. By decreasing the production of reactive oxygen species (ROS) and free radicals, it may also prevent ROS-induced activation of nuclear factor-kappa B (NF-kB) transcription factor and the production of inflammatory cytokines such as interleukin-1beta (IL-1b), IL-6 and tumor necrosis factor-alpha (TNF-a). In addition, astaxanthin may inhibit cyclooxygenase-1 (COX-1) and nitric oxide (NO) activities, thereby reducing inflammation. Oxidative stress and inflammation play key roles in the pathogenesis of many diseases, including cardiovascular, neurological, autoimmune and neoplastic diseases.
1 2D Structure

Astaxanthin

2 Identification
2.1 Computed Descriptors
2.1.1 IUPAC Name
(6S)-6-hydroxy-3-[(1E,3E,5E,7E,9E,11E,13E,15E,17E)-18-[(4S)-4-hydroxy-2,6,6-trimethyl-3-oxocyclohexen-1-yl]-3,7,12,16-tetramethyloctadeca-1,3,5,7,9,11,13,15,17-nonaenyl]-2,4,4-trimethylcyclohex-2-en-1-one
2.1.2 InChI
InChI=1S/C40H52O4/c1-27(17-13-19-29(3)21-23-33-31(5)37(43)35(41)25-39(33,7)8)15-11-12-16-28(2)18-14-20-30(4)22-24-34-32(6)38(44)36(42)26-40(34,9)10/h11-24,35-36,41-42H,25-26H2,1-10H3/b12-11+,17-13+,18-14+,23-21+,24-22+,27-15+,28-16+,29-19+,30-20+/t35-,36-/m0/s1
2.1.3 InChI Key
MQZIGYBFDRPAKN-UWFIBFSHSA-N
2.1.4 Canonical SMILES
CC1=C(C(CC(C1=O)O)(C)C)C=CC(=CC=CC(=CC=CC=C(C)C=CC=C(C)C=CC2=C(C(=O)C(CC2(C)C)O)C)C)C
2.1.5 Isomeric SMILES
CC1=C(C(C[C@@H](C1=O)O)(C)C)/C=C/C(=C/C=C/C(=C/C=C/C=C(/C=C/C=C(/C=C/C2=C(C(=O)[C@H](CC2(C)C)O)C)\C)\C)/C)/C
2.2 Other Identifiers
2.2.1 UNII
8XPW32PR7I
2.3 Synonyms
2.3.1 MeSH Synonyms

1. Astaxanthine

2. E-astaxanthin

2.3.2 Depositor-Supplied Synonyms

1. 472-61-7

2. Ovoester

3. All-trans-astaxanthin

4. Astareal

5. 3,3'-dihydroxy-beta,beta-carotene-4,4'-dione

6. Astaxanthine

7. Bioastin

8. (3s,3's)-astaxanthin

9. Trans-astaxanthin

10. Astaxanthin, (3s,3's)-

11. (3s,3's)-all-trans-astaxanthin

12. 8xpw32pr7i

13. 3,3'-dihydroxy-beta-carotene-4,4'-dione

14. (3s,3's)-3,3'-dihydroxy-beta,beta-carotene-4,4'-dione

15. Chebi:40968

16. Nsc-635689

17. (6s,6's)-3,3'-((1e,3e,5e,7e,9e,11e,13e,15e,17e)-3,7,12,16-tetramethyloctadeca-1,3,5,7,9,11,13,15,17-nonaene-1,18-diyl)bis(6-hydroxy-2,4,4-trimethylcyclohex-2-enone)

18. .beta.,.beta.-carotene-4,4'-dione, 3,3'-dihydroxy-, (3s,3's)-

19. Algae Haematococcus Pluvialis

20. Natupink

21. Astaxin

22. Carophyll Pink

23. Lucantin Pink

24. Bioastin Oleoresin

25. (6s)-6-hydroxy-3-[(1e,3e,5e,7e,9e,11e,13e,15e,17e)-18-[(4s)-4-hydroxy-2,6,6-trimethyl-3-oxo-1-cyclohexenyl]-3,7,12,16-tetramethyloctadeca-1,3,5,7,9,11,13,15,17-nonaenyl]-2,4,4-trimethyl-1-cyclohex-2-enone

26. Astaxanthin (6ci)

27. Astaxanthin, All-trans-

28. Unii-8xpw32pr7i

29. Astaxantin

30. Ccris 7118

31. Hsdb 7468

32. (6s)-6-hydroxy-3-[(1e,3e,5e,7e,9e,11e,13e,15e,17e)-18-[(4s)-4-hydroxy-2,6,6-trimethyl-3-oxocyclohexen-1-yl]-3,7,12,16-tetramethyloctadeca-1,3,5,7,9,11,13,15,17-nonaenyl]-2,4,4-trimethylcyclohex-2-en-1

33. Einecs 207-451-4

34. Nsc 635689

35. 3s,3's-astaxanthin

36. Astaxanthin, All-trans-, (3s,3's)-

37. Astaxanthin, 5% Active

38. Astaxanthin [mi]

39. B-carotene-4,4'-dione

40. Astaxanthin [hsdb]

41. Astaxanthin [inci]

42. Coriolus Versicolor Extract

43. Astaxanthin [vandf]

44. Astaxanthin [mart.]

45. Schembl20047

46. Astaxanthin [usp-rs]

47. Astaxanthin [who-dd]

48. E161j

49. Chembl1255871

50. Astaxanthin, >=98% (hplc)

51. E 161j

52. Dtxsid00893777

53. All-trans-(3s,3's)-astaxanthin

54. 3,3'-dihydroxy-beta,beta-carotene-4,4'-dione, (3s,3's)-

55. Hms3885c08

56. Bcp05821

57. Hy-b2163

58. Lmpr01070263

59. Mfcd00672621

60. S3834

61. Akos015841055

62. Akos015895756

63. Zinc100042059

64. Ac-8760

65. Bcp9000329

66. Ccg-270185

67. Db06543

68. 3,3'-dihydroxy-ss-carotene-4,4'-dione

69. All-trans-astaxanthin, Analytical Standard

70. As-14095

71. Astaxanthin 10 Microg/ml In Acetonitrile

72. Cs-0020413

73. C08580

74. M01303

75. 3,3'-dihydroxy-4,4'-diketo-beta,beta-carotene

76. 472a617

77. A827177

78. Q413740

79. Q-200655

80. 3,3'-dihydroxy-4,4'-diketo-.beta.-carotene

81. All-trans-3,3'-dihydroxy-b-carotene-4,4'-dione (8ci)

82. .beta.-carotene-4,4'-dione, 3,3'-dihydroxy-, All-trans-

83. 3-(4,6-dimethyl-2-oxo-2h-pyrimidin-1-yl)-propionicacid

84. All-trans-3,3'-dihydroxy-beta-carotene-4,4'-dione (8ci)

85. Astaxanthin (constituent Of Astaxanthin Esters) [dsc]

86. 3,3'-dihydroxy-.beta.,.beta.-carotene-4,4'-dione, (3s,3's)-

87. (3s,3's)-3,3'-dihydroxy-.beta.,.beta.-carotene-4,4'-dione

88. Astaxanthin. Short Expiry Date Due To Chemical Nature Of Component(s)

89. 3,3'-dihydroxy-beta,beta-carotene-4,4'-dione;(s)-6-hydroxy-3-((1e,3e,5e,7e,9e,11e,13e,15e,17e)-18-((s)-4-hydroxy-2,6,6-trimethyl-3-oxocyclohex-1-enyl)-3,7,12,16-tetramethyloctadeca-1,3,5,7,9,11,13,15,17-nonaenyl)-2,4,4-trimethylcyclohex-2-enone;

2.4 Create Date
2005-06-24
3 Chemical and Physical Properties
Molecular Weight 596.8 g/mol
Molecular Formula C40H52O4
XLogP310.3
Hydrogen Bond Donor Count2
Hydrogen Bond Acceptor Count4
Rotatable Bond Count10
Exact Mass596.38656014 g/mol
Monoisotopic Mass596.38656014 g/mol
Topological Polar Surface Area74.6 Ų
Heavy Atom Count44
Formal Charge0
Complexity1340
Isotope Atom Count0
Defined Atom Stereocenter Count2
Undefined Atom Stereocenter Count0
Defined Bond Stereocenter Count9
Undefined Bond Stereocenter Count0
Covalently Bonded Unit Count1
4 Drug and Medication Information
4.1 Therapeutic Uses

For the current study, it was hypothesized that oral Cardax /disodium disuccinate astaxanthin/ administration would inhibit oxidative damage of multiple relevant biological targets in a representative, well-characterized murine peritoneal inflammation model. A previously developed mass spectrometry-based (LC/ESI/MS/MS) approach was used to interrogate multiple distinct pathways of oxidation in a black mouse (C57/BL6) model system. In vivo markers of oxidant stress from peritoneal lavage samples (supernatants) were evaluated in mice on day eight (8) after treatment with either Cardax or vehicle (lipophilic emulsion without drug) orally by gavage at 500 mg/kg once per day for seven (7) days at five (5) time points: (1) baseline prior to treatment (t=0); (2) 16 h following intraperitoneal (i.p.) injection with thioglycollate to elicit a neutrophilic infiltrate; (3) 4 h following i.p. injection of yeast cell wall (zymosan; t=16 h/4 h thioglycollate+zymosan); (4) 72 h following i.p. injection with thioglycollate to elicit monocyte/macrophage infiltration; and (5) 72 h/4 h thioglycollate+zymosan. A statistically significant sparing effect on the arachidonic acid (AA) and linoleic acid (LA) substrates was observed at time points two and five. When normalized to the concentration of the oxidative substrates, statistically significant reductions of 8-isoprostane-F(2alpha) (8-iso-F(2alpha)) at time point three (maximal neutrophil recruitment/activation), and 5-HETE, 5-oxo-EET, 11-HETE, 9-HODE, and PGF(2alpha) at time point five (maximal monocyte/macrophage recruitment/activation) were observed. Subsequently, the direct interaction of the optically inactive stereoisomer of Cardax (meso-dAST) with human 5-lipoxygenase (5-LOX) was evaluated in vitro with circular dichroism (CD) and electronic absorption (UV/Vis) spectroscopy, and subsequent molecular docking calculations were made using mammalian 15-LOX as a surrogate (for which XRC data has been reported). The results suggested that the meso-compound was capable of interaction with, and binding to, the solvent-exposed surface of the enzyme. These preliminary studies provide the foundation for more detailed evaluation of the therapeutic effects of this compound on the 5-LOX enzyme, important in chronic diseases such as atherosclerosis, asthma, and prostate cancer in humans. /Disodium disuccinate astaxanthin/

PMID:16466747 Lockwood SF et al; Life Sci 79 (2): 162-74 (2006)


The composition of atherosclerotic plaques, not just macroscopical lesion size, has been implicated in their susceptibility to rupture and the risk of thrombus formation. By focusing on the quality of lipids, macrophages, apoptosis, collagen, metalloproteinase expression and plaque integrity, we evaluated the possible anti-atherosclerotic effect of the antioxidants alpha-tocopherol and astaxanthin in Watanabe heritable hyperlipidemic (WHHL) rabbits. Thirty-one WHHL rabbits were divided into three groups and were fed a standard diet, as controls (N =10), or a standard diet with the addition of 500 mg alpha-tocopherol per kg feed (N =11) or 100 mg astaxanthin per kg feed (N =10) for 24 weeks. We found that both antioxidants, particularly astaxanthin, significantly decreased macrophage infiltration in the plaques although they did not affect lipid accumulation. All lesions in the astaxanthin-treated rabbits were classified as early plaques according to the distribution of collagen and smooth muscle cells. Both antioxidants also improved plaque stability and significantly diminished apoptosis, which mainly occurred in macrophages, matrix metalloproteinase three expressions and plaque ruptures. Although neither antioxidant altered the positive correlations between the lesion size and lipid accumulation, the lesion size and apoptosis were only positively correlated in the control group. Astaxanthin and alpha-tocopherol may improve plaque stability by decreasing macrophage infiltration and apoptosis in this atherosclerotic setting. Apoptosis reduction by alpha-tocopherol and astaxanthin may be a new anti-atherogenic property of these antioxidants.

PMID:15522274 Li W et al; J Mol Cell Cardiol 37 (5): 969-78 (2004)


Exptl Ther: Astaxanthin, a carotenoid without vitamin A activity, may exert antitumor activity through the enhancement of immune responses. Here, we determined the effects of dietary astaxanthin on tumor growth and tumor immunity against transplantable methylcholanthrene-induced fibrosarcoma (Meth-A tumor) cells. These tumor cells express a tumor antigen that induces T cell-mediated immune responses in syngenic mice. BALB/c mice were fed astaxanthin (0.02%, 40 micrograms/kg body wt/day in a beadlet form) mixed in a chemically defined diet starting zero, one, and three weeks before subcutaneous inoculation with tumor cells (3 x 10(5) cells, 2 times the minimal tumorigenic dose). Three weeks after inoculation, tumor size and weight were determined. We also determined cytotoxic T lymphocyte (CTL) activity and interferon-gamma (IFN-gamma) production by tumor-draining lymph node (TDLN) and spleen cells by restimulating cells with Meth-A tumor cells in culture. The astaxanthin-fed mice had significantly lower tumor size and weight than controls when supplementation was started one and three weeks before tumor inoculation. This antitumor activity was paralleled with higher CTL activity and IFN-gamma production by TDLN and spleen cells in the astaxanthin-fed mice. CTL activity by TDLN cells was highest in mice fed astaxanthin for three weeks before inoculation. When the astaxanthin-supplemented diet was started at the same time as tumor inoculation, none of these parameters were altered by dietary astaxanthin, except IFN-gamma production by spleen cells. Total serum astaxanthin concentrations were approximately 1.2 mumol/l when mice were fed astaxanthin (0.02%) for four weeks and appeared to increase in correlation with the length of astaxanthin supplementation. Our results indicate that dietary astaxanthin suppressed Meth-A tumor cell growth and stimulated immunity against Meth-A tumor antigen.

PMID:10798217 Jyonouchi H et al; Nutr Cancer 36 (1): 59-65 (2000)


Exptl Ther: In the current study, the improved oral bioavailability of a synthetic astaxanthin derivative (Cardax; disodium disuccinate astaxanthin) was utilized to evaluate its potential effects as a cardioprotective agent after 7-day subchronic oral administration as a feed supplement to Sprague-Dawley rats. Animals received one of two concentrations of Cardax in feed (0.1 and 0.4%; approximately 125 and 500 mg/kg/day, respectively) or control feed without drug for 7 days prior to the infarct study carried out on day 8. Thirty minutes of occlusion of the left anterior descending (LAD) coronary artery was followed by 2 hr of reperfusion prior to sacrifice, a regimen which resulted in a mean infarct size (IS) as a percentage (%) of the area at risk (AAR; IS/AAR,%) of 61 + or - 1.8%. The AAR was quantified by Patent blue dye injection, and IS was determined by triphenyltetrazolium chloride (TTC) staining. Cardax at 0.1 and 0.4% in feed for 7 days resulted in a significant mean reduction in IS/AAR,% to 45 + or - 2.0% (26% salvage) and 39 + or - 1.5% (36% salvage), respectively. Myocardial levels of free astaxanthin achieved after 7-day supplementation at each of the two concentrations (400 + or - 65 nM and 1634 + or - 90 nM, respectively) demonstrated excellent solid-tissue target organ loading after oral supplementation. Parallel trends in reduction of plasma levels of multiple lipid peroxidation products with disodium disuccinate astaxanthin supplementation were observed, consistent with the documented in vitro antioxidant mechanism of action. These results extend the potential utility of this compound for cardioprotection to the elective human cardiovascular patient population, for which 7-day oral pre-treatment (as with statins) provides significant reductions in induced periprocedural infarct size. /Disodium disuccinate astaxanthin/

PMID:16444582 Gross GJ et al; : Mol Cell Biochem 283 (1-2): 23-30 (2006)


For more Therapeutic Uses (Complete) data for ASTAXANTHINE (7 total), please visit the HSDB record page.


4.2 Drug Indication

Investigated for use/treatment in eye disorders/infections, cancer/tumors (unspecified), and asthma.


5 Pharmacology and Biochemistry
5.1 Absorption, Distribution and Excretion

Apparent astaxanthin (3,3'-dihydroxy-beta,beta-carotene-4,4'-dione) digestibility coefficients (ADC) and carotenoid compositions of the muscle, liver, whole kidney and plasma were compared in Atlantic salmon (Salmo salar) and Atlantic halibut (Hippoglossus hippoglossus) fed a diet supplemented with 66 mg astaxanthin kg(-1) dry matter for 112 days. The astaxanthin source consisted of 75% all-E-, 3% 9Z- and 22% 13Z-astaxanthin, of (3R,3'R)-, (3R,3'S; meso)-, and (3S,3'S)-astaxanthin in a 1:2:1 ratio. The ADC of astaxanthin was significantly higher in Atlantic halibut than in Atlantic salmon after 56 and 112 days of feeding (P < 0.05). The ADC of all-E-astaxanthin was significantly higher than ADC of 9Z-astaxanthin (P < 0.05). Considerably more carotenoids were present in all plasma and tissue samples of salmon than in halibut. Retention of astaxanthin in salmon muscle was 3.9% in salmon and 0 in halibut. All-E-astaxanthin accumulated selectively in the muscle of salmon, and in plasma of salmon and halibut compared with diet. 13Z-astaxanthin accumulated selectively in liver and whole kidney of salmon and halibut, when compared with plasma. A reductive pathway for astaxanthin metabolism in halibut similar to that of salmon was shown by the presence of 3',4'-cis and trans glycolic isomers of idoxanthin (3,3',4'-trihydroxy-beta,beta-carotene-4'-one) in plasma, liver and whole kidney. In conclusion, the higher ADC of astaxanthin in halibut than Atlantic salmon may be explained by lower feed intake in halibut, and the lower retention of astaxanthin by a higher capacity to transform astaxanthin metabolically.

PMID:11126773 Bjerkeng B, Berge GM; Comp Biochem Physiol B Biochem Mol Biol 127 (3): 423-32 (2000)


The present studies were performed to investigate the metabolism of astaxanthin (Ax) in Atlantic salmon, especially in the liver of salmon. The investigations were undertaken in vivo salmon that were fed a diet containing 60 ppm 15, 15' (14)C-labelled Ax prior to sacrifice. The samples of blood, bile, liver, gastrointestinal tract and contents, muscle, skin, remaining carcass and feces were taken for scintillation counting. The highest radioactivity (71.36%) of (14)C-labelled Ax was found in the gastrointestinal contents and feces, 7.13% in the bile and 10.68% in the samples of liver, muscle, and skin at the end of the experiments. The metabolites of (14)C-labelled Ax were extracted from the bile of the salmon and analyzed using thin-layer chromatography (TLC) and high performance liquid chromatography (HPLC). Predominant (14)C-labelled Ax and its cis-isomers were found and no conjugation of (14)C-labelled Ax was observed. These results indicate that (14)C-labelled Ax was not conjugated into larger colorless compound in Atlantic salmon liver.

PMID:15039990 Xu Y, Ding Z; J Exp Zoolog A Comp Exp Biol 301 (4):317-23 (2004)


5.2 Metabolism/Metabolites

One force-fed meal containing labelled (14)C-astaxanthin and (3)H-canthaxanthin or (3)H-zeaxanthin was given to eight mature female rainbow trout. Ninety-six hours after the test meal ingestion, trout were killed and liver, skin, muscle and ovaries were dissected out. Astaxanthin accumulated slightly more in muscle than canthaxanthin but in all tissues astaxanthin and canthaxanthin were very significantly more concentrated than zeaxanthin. (3)H-zeaxanthin metabolites were found only in the liver, whereas (14)C-phoenicoxanthin was the only metabolic pigment from (14)C-astaxanthin detected and was found in all investigated tissues. (3)H-astaxanthin was found in the liver of all trout indicating that (3)H-canthaxanthin and (3)H-zeaxanthin were astaxanthin precursors, and that salmonids probably possess carotenoid oxidative pathways unknown until now. Labelled retinol1 and retinol2 were detected only in the liver and (3)H-zeaxanthin was largely the predominant precursor of these two vitamin A forms.

PMID:1526135 Guillou A et al; 1: Comp Biochem Physiol B 102 (1): 61-5 (1992)


The effects of feed intake, growth rate and temperature (8 and 12 degrees C) on apparent digestibility coefficients (ADC), blood uptake of individual astaxanthin E/Z isomers and metabolism of astaxanthin (3,3'-dihydroxy-beta,beta-carotene-4,4'-dione) were determined in Atlantic salmon. Accumulation of idoxanthin (3,4,3'-trihydroxy-beta,beta-carotene-4-one) in plasma was used to indicate metabolic transformation of astaxanthin.

PMID:16242366 Ytrestoyl T et al; Comp Biochem Physiol B Biochem Mol Biol 142 (4): 445-55 (2005)


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Astaxanthin

About the Company : Hunan Bitian Technology Co.,Ltd is dedicate to R&D, Manufacturing and Distribution of Natural Ingredients, Natural Colors, Natrual Fruit & Vegetable Powder, Cosmetic Materials and ...

Hunan Bitian Technology Co.,Ltd is dedicate to R&D, Manufacturing and Distribution of Natural Ingredients, Natural Colors, Natrual Fruit & Vegetable Powder, Cosmetic Materials and Equipments for clients in the field of food, feed, pharmaceutical and cosmetic. Bitian Team is made up of a group of professionals with considerable experience and achievements in the natrual ingredients industry. It consists of experts trained in natural ingredients sciences, industry managements and internationally trade experienced professionals.
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Astaxanthin

About the Company : Newgen Biotech® USA provides a global platform for accessing the highest quality ingredients and blends. We source our products from manufacturing sites that adhere to ISO9001, IS...

Newgen Biotech® USA provides a global platform for accessing the highest quality ingredients and blends. We source our products from manufacturing sites that adhere to ISO9001, ISO22000, HACCP, HARPC, and GMPs for NSF GMP for US Food, as well as pharmaceutical GMP standards for international companies with different standards from the US. As a well-established, contract manufacturer and distributor, we serve various industries such as Food & Beverage, Human Nutrition, and Cosmetics. Our key manufacturer's products and processes hold over 30 patents, and many of our offerings are certified HALAL, KOSHER, and comply with REACH standards.
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07

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Astaxanthin

About the Company : Summit ingredient Co.,Ltd is a leading and rapidly growing company wh ich engages in the production, development, source and sale of botanical extracts . Summit located in Shaanxi ...

Summit ingredient Co.,Ltd is a leading and rapidly growing company wh ich engages in the production, development, source and sale of botanical extracts . Summit located in Shaanxi City, famous for its natural resources which is very beautiful and rich . Summit ingredient Co.,Ltd have strong R&D team with advanced quality control and production technology. Summit ingredient continues make innovations with the specialty ingredients that serve the dietary supplement, functional food, cosmetic industries for customers all over the world.
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08

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Astaxanthin

About the Company : Zenfold Sustainable Technologies (ZST) is a specialty and fine chemicals company, established in 2021. Our advanced technological capabilities and state-of-the-art infrastructure a...

Zenfold Sustainable Technologies (ZST) is a specialty and fine chemicals company, established in 2021. Our advanced technological capabilities and state-of-the-art infrastructure allow us to provide the entire gamut of services involved in manufacturing and contract research — from process development to scaling up. We offer a wide range of APIs and intermediates to customers across industries. We differentiate ourselves in the market with our unique hybridized approach to manufacturing, built on our three core technological platforms: Synthetic Biology, Biotransformation, and Green Chemistry.
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API Reference Price

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L","supplierCountry":"CHINA","foreign_port":"BEIJING","customer":"SREDSTVA REGIONALE CHEMIE PRIVATE LIMITED","customerCountry":"INDIA","quantity":"50.00","actualQuantity":"50","unit":"KGS","unitRateFc":"220","totalValueFC":"11113","currency":"USD","unitRateINR":"18579","date":"17-Jul-2024","totalValueINR":"928950","totalValueInUsd":"11113","indian_port":"Bombay Air","hs_no":"29161590","bill_no":"4553279","productDescription":"API","marketType":"REGULATED MARKET","country":"CHINA","selfForZScoreResived":"Pharma Grade","supplierPort":"BEIJING","supplierAddress":"TD., A-804, HUIHANG PLAZA, HANG TUOROAD, CHANG\\'AN DISTRICT, 710000 XI\\'AN, CN","customerAddress":"SAVGAN HEIGHT UNIT B 1003"}]
30-Jun-2021
16-Sep-2024
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USP

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01

USP

U.S.A
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Astaxanthin (Synthetic) (3 x 30 mg)

CAS Number : 7542-45-2

Quantity Per Vial :

Price ($) : 225

Catalog Number : 1044200

Current Lot : R036P0

Previous Lot : R006L0 (31-JUL-2017)

NDC Code :

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ABOUT THIS PAGE

Astaxanthin Manufacturers

A Astaxanthin manufacturer is defined as any person or entity involved in the manufacture, preparation, processing, compounding or propagation of Astaxanthin, including repackagers and relabelers. The FDA regulates Astaxanthin manufacturers to ensure that their products comply with relevant laws and regulations and are safe and effective to use. Astaxanthin API Manufacturers are required to adhere to Good Manufacturing Practices (GMP) to ensure that their products are consistently manufactured to meet established quality criteria.

click here to find a list of Astaxanthin manufacturers with USDMF, JDMF, KDMF, CEP, GMP, COA and API Price related information on PhamaCompass.

Astaxanthin Suppliers

A Astaxanthin supplier is an individual or a company that provides Astaxanthin active pharmaceutical ingredient (API) or Astaxanthin finished formulations upon request. The Astaxanthin suppliers may include Astaxanthin API manufacturers, exporters, distributors and traders.

click here to find a list of Astaxanthin suppliers with USDMF, JDMF, KDMF, CEP, GMP, COA and API Price related information on PharmaCompass.

Astaxanthin GMP

Astaxanthin Active pharmaceutical ingredient (API) is produced in GMP-certified manufacturing facility.

GMP stands for Good Manufacturing Practices, which is a system used in the pharmaceutical industry to make sure that goods are regularly produced and monitored in accordance with quality standards. The FDA’s current Good Manufacturing Practices requirements are referred to as cGMP or current GMP which indicates that the company follows the most recent GMP specifications. The World Health Organization (WHO) has its own set of GMP guidelines, called the WHO GMP. Different countries can also set their own guidelines for GMP like China (Chinese GMP) or the EU (EU GMP).

PharmaCompass offers a list of Astaxanthin GMP manufacturers, exporters & distributors, which can be sorted by USDMF, JDMF, KDMF, CEP (COS), WC, API price, and more, enabling you to easily find the right Astaxanthin GMP manufacturer or Astaxanthin GMP API supplier for your needs.

Astaxanthin CoA

A Astaxanthin CoA (Certificate of Analysis) is a formal document that attests to Astaxanthin's compliance with Astaxanthin specifications and serves as a tool for batch-level quality control.

Astaxanthin CoA mostly includes findings from lab analyses of a specific batch. For each Astaxanthin CoA document that a company creates, the USFDA specifies specific requirements, such as supplier information, material identification, transportation data, evidence of conformity and signature data.

Astaxanthin may be tested according to a variety of international standards, such as European Pharmacopoeia (Astaxanthin EP), Astaxanthin JP (Japanese Pharmacopeia) and the US Pharmacopoeia (Astaxanthin USP).

Inform the supplier about your product requirements, specifying if you need a product with particular monograph like EP (Ph. Eur.), USP, JP, BP, or any other quality. In addition, clarify whether you need hydrochloride (HCl), anhydricum, base, micronisatum or a specific level of purity. To find reputable suppliers, utilize the filters and select those certified by GMP, FDA, or any other certification as per your requirement.
For your convenience, we have listed synonyms and CAS numbers to help you find the best supplier. The use of synonyms and CAS numbers can be helpful in identifying potential suppliers, but it is crucial to note that they might not always indicate the exact same product. It is important to confirm the product details with the supplier before making a purchase to ensure that it meets your requirements.
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