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Finasteride

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ACTIVE PHARMA INGREDIENTS

30 API Suppliers, 12 USDMF, 11 CEP/COS, 6 JDMF, 8 EU WC, 32 KDMF, 20 NDC API, 20 Listed Suppliers, $ API Reference Price

30 API Suppliers
12 USDMF
11 CEP/COS
6 JDMF
8 EU WC
32 KDMF
20 NDC API
20 Listed Suppliers
$ API Reference Price

DEVELOPMENTS

7 Drugs in Development

7 Drugs in Development

FINISHED DOSAGE FORMULATIONS

247 FDF Dossiers, 24 FDA Orange Book, 135 Europe, 25 Canada, 20 Australia, 10 South Africa, 33 Listed Dossiers

247 FDF Dossiers
24 FDA Orange Book
135 Europe
25 Canada
20 Australia
10 South Africa
33 Listed Dossiers

DRUG PRODUCT COMPOSITIONS

TABLET;ORAL - 5MG, TABLET;ORAL - 1MG

drugProductComposition
TABLET;ORAL - 5MG
TABLET;ORAL - 1MG

RELATED EXCIPIENT COMPANIES

4 SPI Pharma, 2 Seqens, 4 DKSH, 7 Faran Shimi Pharmaceutical, 4 Boai NKY Pharmaceuticals Ltd, 13 Gangwal Healthcare, 6 Nanjing Well Pharmaceutical, 1 ACG Worldwide, 9 Actylis, 1 Aeon Procare, 8 Anhui Sunhere Pharmaceutical Excipients Co.,Ltd, 25 BASF, 1 Clariant, 10 Corel Pharma Chem, 7 DFE Pharma, 2 Finar, 10 Gangwal Chemicals, 5 Ideal Cures Pvt Ltd, 4 Ingredion Pharma Solutions, 37 Kerry, 3 Kima Chemical, 6 Lonza Capsugel, 12 Microlex e.U, 1 Nanjing Bold Chemical, 2 Nomisma Healthcare, 2 Novo Excipients, 3 Pharmatrans-Sanaq, 8 Pluviaendo, 4 Prachin Chemical, 1 Qianhao Chemical (Hebei) Co., Ltd, 1 Qualicaps, 18 Roquette, 14 Seppic, 6 Shanghai Shenmei Pharmaceutical Technology Co., Ltd, 1 Shanghai Welltone Material Technology Co., Ltd, 12 Sigachi Industries, 2 The Dow Chemical Company, 1 Ulanqab Kema New Material, 1 Valens Pharmachem, 4 Vasa Pharmachem

EXCIPIENTS BY APPLICATIONS

71 Fillers, Diluents & Binders, 49 Coating Systems & Additives, 34 Direct Compression, 30 Controlled & Modified Release, 29 Disintegrants & Superdisintegrants, 25 Solubilizers, 24 Granulation, 23 Thickeners and Stabilizers, 20 Topical, 19 Co-Processed Excipients, 18 Lubricants & Glidants, 17 Parenteral, 16 Film Formers & Plasticizers, 9 Taste Masking, 9 Emulsifying Agents, 6 API Stability Enhancers, 6 Chewable & Orodispersible Aids, 5 Empty Capsules, 5 Coloring Agents, 5 Rheology Modifiers, 4 Vegetarian Capsules, 4 Surfactant & Foaming Agents

DIGITAL CONTENT

2 DATA COMPILATION #PharmaFlow, 19 NEWS #PharmaBuzz

media
2 DATA COMPILATION #PharmaFlow
19 NEWS #PharmaBuzz

GLOBAL SALES INFORMATION

2 US Medicaid Prescriptions, 122 Finished Drug Prices, 9 Annual Reports

globalSalesInformation
2 US Medicaid Prescriptions
122 Finished Drug Prices
9 Annual Reports

MARKET PLACE

21 APIs, 9 FDF Dossiers

marketPlace
21 APIs
9 FDF Dossiers

PATENTS & EXCLUSIVITIES

7 Health Canada Patents

patents
7 Health Canada Patents

REF. STANDARDS & IMPURITIES

2 EDQM , 1 USP , 1 Others

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2 EDQM
1 USP
1 Others

ANALYTICAL

1 BioAnalytical Methods

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1 BioAnalytical Methods

Synopsis

ACTIVE PHARMA INGREDIENTS

API Suppliers

USDMF

CEP/COS

JDMF

EU WC

KDMF

NDC API

VMF

Listed Suppliers

API REF. PRICE (USD/KG)

$ 3,015

MARKET PLACE

API

FDF

0INTERMEDIATES

FINISHED DOSAGE FORMULATIONS

247

FDF Dossiers

FDA Orange Book

135

Europe

Canada

Australia

South Africa

Listed Dossiers

7DRUGS IN DEVELOPMENT

DRUG PRODUCT COMPOSITIONS

REF. STANDARDS OR IMPURITIES

EDQM

USP

Others

PATENTS & EXCLUSIVITIES

US Patents

US Exclusivities

Health Canada Patents

DIGITAL CONTENT

Data Compilation #PharmaFlow

Stock Recap #PipelineProspector

Weekly News Recap #Phispers

News #PharmaBuzz

GLOBAL SALES INFORMATION

US Medicaid (USD)

112,569,027

Annual Reports (USD Million)

958

Finished Drug Prices

262 RELATED EXCIPIENT COMPANIES

428EXCIPIENTS BY APPLICATIONS

Chemistry

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Also known as: 98319-26-7, Proscar, Propecia, Finastid, Prostide, Chibro-proscar

Molecular Formula

C₂₃H₃₆N₂O₂

Molecular Weight

372.5 g/mol

InChI Key

DBEPLOCGEIEOCV-WSBQPABSSA-N

FDA UNII

57GNO57U7G

An orally active 3-OXO-5-ALPHA-STEROID 4-DEHYDROGENASE inhibitor. It is used as a surgical alternative for treatment of benign PROSTATIC HYPERPLASIA.

Finasteride is a 5-alpha Reductase Inhibitor. The mechanism of action of finasteride is as a 5-alpha Reductase Inhibitor.

1 2D Structure

Finasteride

2 Identification

2.1 Computed Descriptors

2.1.1 IUPAC Name

(1S,3aS,3bS,5aR,9aR,9bS,11aS)-N-tert-butyl-9a,11a-dimethyl-7-oxo-1,2,3,3a,3b,4,5,5a,6,9b,10,11-dodecahydroindeno[5,4-f]quinoline-1-carboxamide

2.1.2 InChI

InChI=1S/C23H36N2O2/c1-21(2,3)25-20(27)17-8-7-15-14-6-9-18-23(5,13-11-19(26)24-18)16(14)10-12-22(15,17)4/h11,13-18H,6-10,12H2,1-5H3,(H,24,26)(H,25,27)/t14-,15-,16-,17+,18+,22-,23+/m0/s1

2.1.3 InChI Key

DBEPLOCGEIEOCV-WSBQPABSSA-N

2.1.4 Canonical SMILES

CC12CCC3C(C1CCC2C(=O)NC(C)(C)C)CCC4C3(C=CC(=O)N4)C

2.1.5 Isomeric SMILES

C[C@]12CC[C@H]3[C@H]([C@@H]1CC[C@@H]2C(=O)NC(C)(C)C)CC[C@@H]4[C@@]3(C=CC(=O)N4)C

2.2 Other Identifiers

2.2.1 UNII

57GNO57U7G

2.3 Synonyms

2.3.1 MeSH Synonyms

1. Chibro Proscar

2. Chibro-proscar

3. Eucoprost

4. Mk 906

5. Mk-906

6. Mk906

7. Propecia

8. Propeshia

9. Proscar

2.3.2 Depositor-Supplied Synonyms

1. 98319-26-7

2. Proscar

3. Propecia

4. Finastid

5. Prostide

6. Chibro-proscar

7. Mk-906

8. Finpecia

9. Finasterida

10. Finasteridum

11. Mk 906

12. (5alpha,17beta)-(1,1-dimethylethyl)-3-oxo-4-azaandrost-1-ene-17-carboxamide

13. (4ar,4bs,6as,7s,9as,9bs,11ar)-n-tert-butyl-4a,6a-dimethyl-2-oxo-2,4a,4b,5,6,6a,7,8,9,9a,9b,10,11,11a-tetradecahydro-1h-indeno[5,4-f]quinoline-7-carboxamide

14. Chembl710

15. Nsc-741485

16. N-tert-butyl-3-oxo-4-aza-5alpha-androst-1-ene-17beta-carboxamide

17. 57gno57u7g

18. Chebi:5062

19. (1s,3as,3bs,5ar,9ar,9bs,11as)-n-tert-butyl-9a,11a-dimethyl-7-oxo-1,2,3,3a,3b,4,5,5a,6,9b,10,11-dodecahydroindeno[5,4-f]quinoline-1-carboxamide

20. (4ar,4bs,6as,7s,9as,9bs,11ar)-n-(tert-butyl)-4a,6a-dimethyl-2-oxo-2,4a,4b,5,6,6a,7,8,9,9a,9b,10,11,11a-tetradecahydro-1h-indeno[5,4-f]quinoline-7-carboxamide

21. Nsc-759318

22. Finasteridum [inn-latin]

23. Finasterida [inn-spanish]

24. Dsstox_cid_625

25. Dsstox_rid_75699

26. Dsstox_gsid_20625

27. (4ar,4bs,6as,7s,9as,9bs,11ar)-n-(1,1-dimethylethyl)-4a,6a-dimethyl-2-oxo-2,4a,4b,5,6,6a,7,8,9,9a,9b,10,11,11a-tetradecahydro-1h-indeno[5,4-f]quinoline-7-carboxamide

28. N-tert-butyl-3-oxo-4-aza-5alpha-androst-1-en-17beta-carboxamide

29. Andozac

30. 4-azaandrost-1-ene-17-carboxamide, N-(1,1-dimethylethyl)-3-oxo-, (5alpha,17beta)-

31. Propecia (tn)

32. Smr000466304

33. Proscar (tn)

34. Mk 0906

35. Mk-0906

36. Ccris 7438

37. Hsdb 6793

38. Mk906

39. Sr-01000759414

40. Brn 4269024

41. Unii-57gno57u7g

42. L-652,931

43. 17beta-(n-tert-butylcarbamoyl)-4-aza-5 Alpha-androst-1-en-3-one

44. Ncgc00016965-01

45. Finasteride [usan:usp:inn:ban]

46. N-(2-methyl-2-propyl)-3-oxo-4-aza-5-alpha-androst-1-ene-17-beta-carboxamide

47. Cas-98319-26-7

48. Mfcd00869737

49. Ym-152

50. Ks-1058

51. Finasteride (proscar)

52. Finasteride [mi]

53. Prestwick0_000717

54. Prestwick1_000717

55. Prestwick2_000717

56. Prestwick3_000717

57. Finasteride [inn]

58. Finasteride [jan]

59. Finasteride [usan]

60. Finasteride [vandf]

61. Schembl5509

62. Finasteride [mart.]

63. Bspbio_000933

64. Finasteride [who-dd]

65. Mls000759404

66. Mls001165768

67. Mls001424046

68. Finasteride (jan/usp/inn)

69. Spbio_002854

70. Bpbio1_001027

71. Gtpl6818

72. Dtxsid3020625

73. N-tert-butyl-3-oxo-4-aza-5.alpha.-androst-1-ene-17.beta.-carboxamide

74. Finasteride [ep Impurity]

75. Finasteride [orange Book]

76. 4-azaandrost-1-ene-17-carboxamide, N-(1,1-dimethylethyl)-3-oxo-, (5.alpha.,17.beta.)-

77. Bcpp000229

78. Finasteride [ep Monograph]

79. Finasteride [usp Impurity]

80. Hms1570o15

81. Hms2051f09

82. Hms2090g22

83. Hms2097o15

84. Hms2235l23

85. Hms3714o15

86. Finasteride [usp Monograph]

87. 4a,6a-dimethyl-2-oxo-2,4a,4b,5,6,6a,7,8,9,9a,9b,10,11,11a-tetradecahydro-1h-indeno[5,4-f]quinoline-7-carboxylic Acid Tert-butylamide

88. Act02599

89. Entadfi Component Finasteride

90. Ex-a1951

91. Zinc3782599

92. Tox21_110717

93. Tox21_201506

94. Tox21_302744

95. Bdbm50334788

96. Nsc741485

97. Nsc757443

98. S1197

99. Akos015894916

100. Tox21_110717_1

101. Bcp9000685

102. Ccg-100937

103. Cs-1767

104. Db01216

105. Finasteride Component Of Entadfi

106. Finasteride, >=98% (hplc), Powder

107. Nc00187

108. Nsc 741485

109. Nsc 759318

110. Nsc-757443

111. Ncgc00093560-05

112. Ncgc00256334-01

113. Ncgc00259057-01

114. 140375-21-9

115. Bf164456

116. Cpd000466304

117. Hy-13635

118. Bcp0726000222

119. Ab00513901

120. D00321

121. Ab00513901-07

122. Ab00513901-08

123. Ab00513901_09

124. Finasteride, Vetranal(tm), Analytical Standard

125. 319f267

126. A845840

127. Q424167

128. Sr-01000759414-4

129. Sr-01000759414-6

130. Brd-k01095011-001-03-5

131. Brd-k01095011-001-15-9

132. Finasteride, British Pharmacopoeia (bp) Reference Standard

133. Finasteride, European Pharmacopoeia (ep) Reference Standard

134. ([1-phenyl-meth-(e)-ylidene]-amino)-aceticacidethylester

135. (17beta-(n-tert-butylcarbamoyl)-4-aza-5alpha-androst-1-en-3-one

136. Finasteride, United States Pharmacopeia (usp) Reference Standard

137. (5?,17?)-n-(1,1-dimethylethyl)-3-oxo-4-azaandrost-1-ene-17-carboxamide

138. 4-azaandrost-1-ene-17-carboxamide,1-dimethylethyl)-3-oxo-, (5.alpha., 17.beta.)-

139. Finasteride For Peak Identification, European Pharmacopoeia (ep) Reference Standard

140. (1s,2r,7r,10s,11s,14s,15s)-n-tert-butyl-2,15-dimethyl-5-oxo-6-azatetracyclo[8.7.0.0^{2,7}.0^{11,15}]heptadec-3-ene-14-carboxamide

141. (1s,3as,3bs,5ar,9ar,9bs,11as)-n-isobutyl-9a,11a-dimethyl-7-oxo-1,2,3,3a,3b,4,5,5a,6,9b,10,11-dodecahydroindeno[5,4-f]quinoline-1-carboxamide

142. (1s,3as,3bs,5ar,9ar,9bs,11as)-n-isobutyl-9a,11a-dimethyl-7-oxo-1,2,3,3a,3b,4,5,5a,6,9b,10,11-dodecahydroindeno[5,4-f]quinoline-1-carboxamide;finasteride

143. (4ar,4bs,6as,7s,9as,9bs,11ar)-4a,6a-dimethyl-2-oxo-2,4a,4b,5,6,6a,7,8,9,9a,9b,10,11,11a-tetradecahydro-1h-indeno[5,4-f]quinoline-7-carboxylic Acid Tert-butylamide

144. (4ar,4bs,6as,9as,9bs,11ar)-4a,6a-dimethyl-2-oxo-2,4a,4b,5,6,6a,7,8,9,9a,9b,10,11,11a-tetradecahydro-1h-indeno[5,4-f]quinoline-7-carboxylic Acid Tert-butylamide

145. (4ar,6as)-4a,6a-dimethyl-2-oxo-2,4a,4b,5,6,6a,7,8,9,9a,9b,10,11,11a-tetradecahydro-1h-indeno[5,4-f]quinoline-7-carboxylic Acid Tert-butylamide

146. (4ar,6as,11ar)-4a,6a-dimethyl-2-oxo-hexadecahydro-indeno[5,4-f]quinoline-7-carboxylic Acid Tert-butylamide

147. (4ar,6as,7s)-4a,6a-dimethyl-2-oxo-2,4a,4b,5,6,6a,7,8,9,9a,9b,10,11,11a-tetradecahydro-1h-indeno[5,4-f]quinoline-7-carboxylic Acid Tert-butylamide

148. (4ar,6as,7s,11ar)-4a,6a-dimethyl-2-oxo-2,4a,4b,5,6,6a,7,8,9,9a,9b,10,11,11a-tetradecahydro-1h-indeno[5,4-f]quinoline-7-carboxylic Acid Tert-butylamide

149. (r)-4a,6a-dimethyl-2-oxo-2,4a,4b,5,6,6a,7,8,9,9a,9b,10,11,11a-tetradecahydro-1h-indeno[5,4-f]quinoline-7-carboxylic Acid Tert-butylamide

150. 1h-indeno[5,4-f]quinoline-7-carboxamide, N-(1,1-dimethylethyl)-2,4a,4b,5,6,6a,7,8,9,9a,9b,10,11,11a-tetradecahydro-4a,6a-dimethyl-2-oxo-, (4ar,4bs,6as,7s,9as,9bs,11ar)-

151. 4a,6a,9a-trimethyl-2-oxo-2,4a,4b,5,6,6a,7,8,9,9a,9b,10,11,11a-tetradecahydro-1h-indeno[5,4-f]quinoline-7-carboxylic Acid (finasteride)

152. 4a,6a-dimethyl-2-oxo-2,4a,4b,5,6,6a,7,8,9,9a,9b,10,11,11a-tetradecahydro-1h-indeno[5,4-f]quinoline-7-carboxylic Acid Tert-butylamide(finasteride)

2.4 Create Date

2005-06-24

3 Chemical and Physical Properties

Molecular Formula	C₂₃H₃₆N₂O₂
Molecular Weight	372.5 g/mol
XLogP3	3
Hydrogen Bond Donor Count	2
Hydrogen Bond Acceptor Count	2
Rotatable Bond Count	2
Exact Mass	372.277678395 g/mol
Monoisotopic Mass	372.277678395 g/mol
Topological Polar Surface Area	58.2 Å²
Heavy Atom Count	27
Formal Charge	0
Complexity	678
Isotope Atom Count	0
Defined Atom Stereocenter Count	7
Undefined Atom Stereocenter Count	0
Defined Bond Stereocenter Count	0
Undefined Bond Stereocenter Count	0
Covalently Bonded Unit Count	1

4 Drug and Medication Information

4.1 Drug Information

1 of 6
Drug Name	Finasteride
PubMed Health	Finasteride (By mouth)
Drug Classes	Alopecia Agent, Benign Prostatic Hypertrophy Agent
Drug Label	Finasteride, USP, a synthetic 4-azasteroid compound, is a specific inhibitor of steroid Type II 5-reductase, an intracellular enzyme that converts the androgen testosterone into 5-dihydrotestosterone (DHT).Finasteride is 4-Azaandrost-1-ene-17-car...
Active Ingredient	Finasteride
Dosage Form	Tablet
Route	oral; Oral
Strength	5mg; 1mg
Market Status	Tentative Approval; Prescription
Company	Watson Labs; Mylan Pharms; Actavis Elizabeth; Teva; Accord Hlthcare; Hetero Labs Ltd Iii; Aurobindo Pharma; Zydus Pharms Usa; Dr Reddys Labs; Gedeon Richter Usa; Sun Pharma Global; Mylan

2 of 6
Drug Name	Propecia
PubMed Health	Finasteride (By mouth)
Drug Classes	Alopecia Agent, Benign Prostatic Hypertrophy Agent
Drug Label	PROPECIA (finasteride) tablets contain finasteride as the active ingredient. Finasteride, a synthetic 4-azasteroid compound, is a specific inhibitor of steroid Type II 5-reductase, an intracellular enzyme that converts the androgen testosterone int...
Active Ingredient	Finasteride
Dosage Form	Tablet
Route	Oral
Strength	1mg
Market Status	Prescription
Company	Merck

3 of 6
Drug Name	Proscar
Drug Label	PROSCAR (finasteride), a synthetic 4-azasteroid compound, is a specific inhibitor of steroid Type II 5-reductase, an intracellular enzyme that converts the androgen testosterone into 5-dihydrotestosterone (DHT).Finasteride is 4-azaandrost-1-ene-1...
Active Ingredient	Finasteride
Dosage Form	Tablet
Route	Oral
Strength	5mg
Market Status	Prescription
Company	Merck

4 of 6
Drug Name	Finasteride
PubMed Health	Finasteride (By mouth)
Drug Classes	Alopecia Agent, Benign Prostatic Hypertrophy Agent
Drug Label	Finasteride, USP, a synthetic 4-azasteroid compound, is a specific inhibitor of steroid Type II 5-reductase, an intracellular enzyme that converts the androgen testosterone into 5-dihydrotestosterone (DHT).Finasteride is 4-Azaandrost-1-ene-17-car...
Active Ingredient	Finasteride
Dosage Form	Tablet
Route	oral; Oral
Strength	5mg; 1mg
Market Status	Tentative Approval; Prescription
Company	Watson Labs; Mylan Pharms; Actavis Elizabeth; Teva; Accord Hlthcare; Hetero Labs Ltd Iii; Aurobindo Pharma; Zydus Pharms Usa; Dr Reddys Labs; Gedeon Richter Usa; Sun Pharma Global; Mylan

5 of 6
Drug Name	Propecia
PubMed Health	Finasteride (By mouth)
Drug Classes	Alopecia Agent, Benign Prostatic Hypertrophy Agent
Drug Label	PROPECIA (finasteride) tablets contain finasteride as the active ingredient. Finasteride, a synthetic 4-azasteroid compound, is a specific inhibitor of steroid Type II 5-reductase, an intracellular enzyme that converts the androgen testosterone int...
Active Ingredient	Finasteride
Dosage Form	Tablet
Route	Oral
Strength	1mg
Market Status	Prescription
Company	Merck

6 of 6
Drug Name	Proscar
Drug Label	PROSCAR (finasteride), a synthetic 4-azasteroid compound, is a specific inhibitor of steroid Type II 5-reductase, an intracellular enzyme that converts the androgen testosterone into 5-dihydrotestosterone (DHT).Finasteride is 4-azaandrost-1-ene-1...
Active Ingredient	Finasteride
Dosage Form	Tablet
Route	Oral
Strength	5mg
Market Status	Prescription
Company	Merck

4.2 Therapeutic Uses

Enzyme Inhibitors

National Library of Medicine's Medical Subject Headings online file (MeSH, 1999)

Treatment of benign prostatic hypertrophy

Budavari, S. (ed.). The Merck Index - Encyclopedia of Chemicals, Drugs and Biologicals. Rahway, NJ: Merck and Co., Inc., 1989., p. 1250

Antiandrogen therapy appears to produce a 30 to 40% decrease in the volume of the hyperplastic prostate after 3 to 6 months of therapy. Longer treatment may result in further prostatic regression, although this remains to be seen. Biopsy studies suggest that epithelial regression occurs to a much more significant degree than does stromal regression, but this finding may simply reflect the relatively longer turnover of the stromal cell population. The significant placebo effect of oral medication in patients with benign prostatic hyperplasia makes interpretation of clinical symptomatology and uro-flow data difficult. Analysis of symptom improvement is further complicated by the relatively slow improvement of patients on antiandrogen therapy, in contrast to surgery, in which relief is immediate. In addition to limited stromal involution and inadequate treatment duration, other biologic factors may limit the clinical efficacy of antiandrogen therapy. Most importantly, prostatic involution may not necessarily decrease urethral resistance. In addition, obstruction induced detrusor dysfunction may persist after relief of outflow obstruction in some patients, as it does after surgery. Incomplete antiandrogen action of the compounds, as well as compliance issues, may likewise limit efficacy. Although there are no data to suggest that the 5 alpha-reductase inhibitor finasteride will be more effective than other antiandrogen compounds in the treatment of benign prostatic hyperplasia, preliminary studies suggest that it has less toxicity. If long-term studies validate a modest but significant clinical response rate and preservation of sexual function, then finasteride therapy may well be acceptable to a subgroup of men presenting with the symptoms of benign prostatic hyperplasia.

PMID:1695786 McConnell JD; Urol Clin North Am 17 (3): 661-70 (1990)

4.3 Drug Indication

Finasteride is indicated for the treatment of symptomatic benign prostatic hyperplasia (BPH) in men with an enlarged prostate to improve symptoms, reduce the risk of acute urinary retention, and reduce the risk of the need for surgery including transurethral resection of the prostate (TURP) and prostatectomy. A combination product with [tadalafil] is also used for the symptomatic treatment of BPH for up to 26 weeks. Finasteride is also indicated for the treatment of male pattern hair loss (androgenetic alopecia, hereditary alopecia, or common male baldness) in male patients.

Treatment of androgenetic alopecia

5 Pharmacology and Biochemistry

5.1 Pharmacology

Finasteride is an antiandrogenic compound that works by suppressing the production of serum and intraprostatic dihydrotestosterone (DHT) in men via inhibiting the enzyme responsible for the biosynthesis of DHT. The maximum effect of a rapid reduction in serum DHT concentration is expected to be observed 8 hours following administration of the first dose. In a single man receiving a single oral dose of 5 mg finasteride for up to 4 years, there was a reduction in the serum DHT concentrations by approximately 70% and the median circulating level of testosterone increased by approximately 10-20% within the physiologic range. In a double-blind, placebo-controlled study, finasteride reduced intraprostatic DHT level by 91.4% but finasteride is not expected to decrease the DHT levels to castrate levels since circulating testosterone is also converted to DHT by the type 1 isoenzyme expressed in other tissues. It is expected that DHT levels return to normal within 14 days upon discontinuation of the drug. In a study of male patients with benign prostatic hyperplasia prior to prostatectomy, the treatment with finasteride resulted in an approximate 80% lower DHT content was measured in prostatic tissue removed at surgery compared to placebo. While finasteride reduces the size of the prostate gland by 20%, this may not correlate well with improvement in symptoms. The effects of finasteride are reported to be more pronounced in male patients with enlarged prostates (>25 mL) who are at the greatest risk of disease progression. In phase III clinical studies, oral administration of finasteride in male patients with male pattern hair loss promoted hair growth and prevented further hair loss by 66% and 83% of the subjects, respectively, which lasted during two years' treatment. The incidences of these effects in treatment groups were significantly higher than that of the group receiving a placebo. Following finasteride administration, the levels of DHT in the scalp skin was shown to be reduced by more than 60%, indicating that the DHT found in scalp is derived from both local DHT production and circulating DHT. The effect of finasteride on scalp DHT is likely seen because of its effect on both local follicular DHT levels as well as serum DHT levels.. There is evidence from early clinical observations and controlled studies that finasteride may reduce bleeding of prostatic origin.

5.2 MeSH Pharmacological Classification

Urological Agents

Drugs used in the treatment of urological conditions and diseases such as URINARY INCONTINENCE and URINARY TRACT INFECTIONS. (See all compounds classified as Urological Agents.)

5-alpha Reductase Inhibitors

Drugs that inhibit 3-OXO-5-ALPHA-STEROID 4-DEHYDROGENASE. They are commonly used to reduce the production of DIHYDROTESTOSTERONE. (See all compounds classified as 5-alpha Reductase Inhibitors.)

5.3 FDA Pharmacological Classification

5.3.1 Active Moiety

FINASTERIDE

5.3.2 FDA UNII

57GNO57U7G

5.3.3 Pharmacological Classes

5-alpha Reductase Inhibitors [MoA]; 5-alpha Reductase Inhibitor [EPC]

5.4 ATC Code

G04CB01

S76 | LUXPHARMA | Pharmaceuticals Marketed in Luxembourg | Pharmaceuticals marketed in Luxembourg, as published by d'Gesondheetskeess (CNS, la caisse nationale de sante, www.cns.lu), mapped by name to structures using CompTox by R. Singh et al. (in prep.). List downloaded from https://cns.public.lu/en/legislations/textes-coordonnes/liste-med-comm.html. Dataset DOI:10.5281/zenodo.4587355

D - Dermatologicals

D11 - Other dermatological preparations

D11A - Other dermatological preparations

D11AX - Other dermatologicals

D11AX10 - Finasteride

G - Genito urinary system and sex hormones

G04 - Urologicals

G04C - Drugs used in benign prostatic hypertrophy

G04CB - Testosterone-5-alpha reductase inhibitors

G04CB01 - Finasteride

5.5 Absorption, Distribution and Excretion

Absorption

Finasteride is well absorbed following oral administration and displays a slow accumulation phase after multiple dosing.[lablel] In healthy male subjects receiving oral finasteride, the mean oral bioavailability was 65% for 1 mg finasteride and 63% for 5 mg finasteride, and the values ranged from 26 to 170% for 1 mg dose and from 34 to 108% for 5 mg dose, respectively. It is reported that food intake does not affect the oral bioavailability of the drug. The peak plasma concentrations (Cmax) averaged 37 ng/mL (range, 27-49 ng/mL) and was reached 1-2 hours post administration. The AUC(0-24 hr) was 53 ngxhr/mL (range, 20-154 ngxhr/mL). The plasma concentrations and AUC are reported to be higher in elderly male patients aged 70 years or older.

Route of Elimination

In healthy subjects, about 32-46% of total oral dose of finasteride was excreted in the urine in the form of metabolites while about 51-64% of the dose was excreted in the feces. In patients with renal impairment, the extent of urinary excretion of finasteride is expected to be decreased while the fecal excretion is increased.

Volume of Distribution

The volume of distribution is 76 L at steady state, ranging from 44 to 96 L. Finasteride has been shown to cross the blood brain barrier but does not appear to distribute preferentially to the CSF. It is not known whether finasteride is excreted in human milk.

Clearance

In healthy young subjects (n=15), the mean plasma clearance of finasteride was 165 mL/min with the range between 70 and 279 mL/min.

5.6 Metabolism/Metabolites

Finasteride undergoes extensive hepatic metabolism predominantly mediated by the cytochrome P450 3A4 (CYP3A4) enzyme to form the t-butyl side chain monohydroxylated and monocarboxylic acid metabolites. Theses metabolites retain less than 20% of the pharmacological activity of the parent compound.

Finasteride has known human metabolites that include N-(1-Hydroxy-2-methylpropan-2-yl)-9a,11a-dimethyl-7-oxo-1,2,3,3a,3b,4,5,5a,6,9b,10,11-dodecahydroindeno[5,4-f]quinoline-1-carboxamide.

S73 | METXBIODB | Metabolite Reaction Database from BioTransformer | DOI:10.5281/zenodo.4056560

5.7 Biological Half-Life

In healthy young subjects receiving finasteride, the mean elimination half-life in plasma was 6 hours ranging from 3 to 16 hours. In elderly patients over the age of 70 years, the half-life is prolonged to 8 hours.

5.8 Mechanism of Action

Finasteride acts as a competitive and specific inhibitor of Type II 5-reductase, a nuclear-bound steroid intracellular enzyme primarily located in the prostatic stromal cell that converts the androgen testosterone into the more active metabolite, 5-dihydrotestosterone (DHT). DHT is considered to be the primary androgen playing a role in the development and enlargement of the prostate gland. It serves as the hormonal mediator for the hyperplasia upon accumulation within the prostate gland. DHT displays a higher affinity towards androgen receptors in the prostate gland compared to testosterone and by acting on the androgen receptors, DHT modulates genes that are responsible for cell proliferation. Responsible for the production of DHT together with type I 5-reductase, the type II 5-reductase isozyme is primarily found in the prostate, seminal vesicles, epididymides, and hair follicles as well as liver. Although finasteride is 100-fold more selective for type II 5-reductase than for the type I isoenzyme, chronic treatment with this drug may have some effect on type I 5-reductase, which is predominantly expressed in sebaceous glands of most regions of skin, including the scalp, and liver. It is proposed that the type I 5-reductase and type II 5-reductase is responsible for the production of one-third and two-thirds of circulating DHT, respectively. The mechanism of action of Finasteride is based on its preferential inhibition of Type II 5-reductase through the formation of a stable complex with the enzyme _in vitro_ and _in vivo_. Finasteride works selectively, where it preferentially displays a 100-fold selectivity for the human Type II 5-reductase over type I enzyme. Inhibition of Type II 5-reductase blocks the peripheral conversion of testosterone to DHT, resulting in significant decreases in serum and tissue DHT concentrations, minimal to moderate increase in serum testosterone concentrations, and substantial increases in prostatic testosterone concentrations. As DHT appears to be the principal androgen responsible for stimulation of prostatic growth, a decrease in DHT concentrations will result in a decrease in prostatic volume (approximately 20-30% after 6-24 months of continued therapy). It is suggested that increased levels of DHT can lead to potentiated transcription of prostaglandin D2, which promotes the proliferation of prostate cancer cells. In men with androgenic alopecia, the mechanism of action has not been fully determined, but finasteride has shown to decrease scalp DHT concentration to the levels found in the hairy scalp, reduce serum DHT, increase hair regrowth, and slow hair loss. Another study suggests that finasteride may work to reduce bleeding of prostatic origin by inhibiting vascular endothelial growth factor (VEGF) in the prostate, leading to atrophy and programmed cell death. This may bestow the drug therapeutic benefits in patients idiopathic prostatic bleeding, bleeding during anticoagulation, or bleeding after instrumentation.

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FINASTERIDE USP (FORM III)

GDUFA

DMF Review : N/A

Rev. Date :

Pay. Date :

DMF Number : 16010

Submission : 2002-06-11

Status : Active

Type : II

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Apitoria Pharma Private Ltd

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API Reference Price

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VAISHNODE","city":"AHMEDABAD","supplier":"HUBEI GEDIAN HUMANWELL PHARMACEUTICAL","supplierCountry":"CHINA","foreign_port":"NA","customer":"ZYDUS LIFESCIENCES LIMITED","customerCountry":"INDIA","quantity":"0.50","actualQuantity":"0.5","unit":"KGS","unitRateFc":"2800","totalValueFC":"1405.4","currency":"USD","unitRateINR":"225260","date":"02-Sep-2022","totalValueINR":"112630","totalValueInUsd":"1405.4","indian_port":"BOMBAY AIR","hs_no":"29372900","bill_no":"2275495","productDescription":"API","marketType":"REGULATED MARKET","country":"CHINA","selfForZScoreResived":"Pharma Grade","supplierPort":"NA","supplierAddress":"","customerAddress":"ZYDUS CORPORATE PARK, SCHEME NO 63, KHORAJ (GANDHINAGAR), NR. VAISHNODE"},{"dataSource":"API Import","activeIngredients":"","year":"2023","qtr":"Q3","strtotime":1692210600,"product":"FINASTERIDE USP\/IP","address":"108, MARINE CHEMBERS, 43, NEW ,MARINE LINES, BEHIND SNDT COLLEGE, ,","city":"Mumbai","supplier":"WUHAN JIULONG PHARMACEUTICAL CO LIMITED","supplierCountry":"CHINA","foreign_port":"NA","customer":"B M CHEMIE","customerCountry":"INDIA","quantity":"100.00","actualQuantity":"100","unit":"KGS","unitRateFc":"1350","totalValueFC":"136217.3","currency":"USD","unitRateINR":"111709.1","date":"17-Aug-2023","totalValueINR":"11283742.8","totalValueInUsd":"136217.3","indian_port":"SAHAR AIR","hs_no":"29379090","bill_no":"0","productDescription":"API","marketType":"REGULATED MARKET","country":"CHINA","selfForZScoreResived":"Pharma Grade","supplierPort":"NA","supplierAddress":"","customerAddress":"108, MARINE CHEMBERS, 43, NEW ,MARINE LINES, BEHIND SNDT COLLEGE, ,"},{"dataSource":"API Import","activeIngredients":"","year":"2024","qtr":"Q2","strtotime":1716402600,"product":"FINASTERIDE(QTY:5 X 200 MG, VALUE: USD 273\/EACH) (FOC)","address":"P.NO. D6 D8, SY.NO. 234\/2,234\/3,","city":"TURKAPALLY(V),SHAMEERPET, HYDERABAD","supplier":"USP","supplierCountry":"INDIA","foreign_port":"WASHINGTON, DULLES I","customer":"UNITED STATES PHARMACOPEIA - INDIA PRIVATE LIMITED","customerCountry":"INDIA","quantity":"0.00","actualQuantity":"0.001","unit":"KGS","unitRateFc":"1365000","totalValueFC":"1410.1","currency":"USD","unitRateINR":"117540640","date":"23-May-2024","totalValueINR":"117540.64","totalValueInUsd":"1410.1","indian_port":"Hyderabad Air","hs_no":"29372900","bill_no":"3620237","productDescription":"Re-Import","marketType":"REGULATED MARKET","country":"INDIA","selfForZScoreResived":"Pharma Grade","supplierPort":"WASHINGTON, DULLES I","supplierAddress":"7135 ENGLISH MUFFIN WAY FREDERICK,MD, US 21704, UNITED STATES OF AMERICA United States","customerAddress":"P.NO. D6 D8, SY.NO. 234\/2,234\/3,"},{"dataSource":"API Import","activeIngredients":"","year":"2024","qtr":"Q4","strtotime":1729276200,"product":"FINASTERIDE IP\/USP","address":"N\/A","city":"","supplier":"WUHAN JULONG PHARMCAEUTICAL CO., LTD","supplierCountry":"CHINA","foreign_port":"WUHAN","customer":"B M CHEMIE PRIVATE LIMITED","customerCountry":"INDIA","quantity":"10.00","actualQuantity":"10","unit":"KGS","unitRateFc":"1350","totalValueFC":"13648.4","currency":"USD","unitRateINR":"114682.5","date":"19-Oct-2024","totalValueINR":"1146825","totalValueInUsd":"13648.4","indian_port":"Bombay Air","hs_no":"29372900","bill_no":"6208533","productDescription":"API","marketType":"REGULATED MARKET","country":"CHINA","selfForZScoreResived":"Pharma Grade","supplierPort":"WUHAN","supplierAddress":"NO.77, OPTICS VALLEY SEVENTH ROAD,E AST LAKE HIGH-TECH DISTRICT, WUHAN 430206 HUBEI CHINA","customerAddress":"N\/A"}]

Number of Transactions

Total Quantity (KGS)

Total Value (USD)

Quantity (KGS) & Unit rate (USD/KGS) over time

API Imports and Exports

Importing Country	Total Quantity (KGS)	Average Price (USD/KGS)	Number of Transactions

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DOSAGE - TABLET;ORAL - 5MG

USFDA APPLICATION NUMBER - 20180

DOSAGE - TABLET;ORAL - 1MG

USFDA APPLICATION NUMBER - 20788

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ABOUT THIS PAGE

Finasteride Manufacturers

A Finasteride manufacturer is defined as any person or entity involved in the manufacture, preparation, processing, compounding or propagation of Finasteride, including repackagers and relabelers. The FDA regulates Finasteride manufacturers to ensure that their products comply with relevant laws and regulations and are safe and effective to use. Finasteride API Manufacturers are required to adhere to Good Manufacturing Practices (GMP) to ensure that their products are consistently manufactured to meet established quality criteria.

click here to find a list of Finasteride manufacturers with USDMF, JDMF, KDMF, CEP, GMP, COA and API Price related information on PhamaCompass.

Finasteride Suppliers

A Finasteride supplier is an individual or a company that provides Finasteride active pharmaceutical ingredient (API) or Finasteride finished formulations upon request. The Finasteride suppliers may include Finasteride API manufacturers, exporters, distributors and traders.

click here to find a list of Finasteride suppliers with USDMF, JDMF, KDMF, CEP, GMP, COA and API Price related information on PharmaCompass.

Finasteride USDMF

A Finasteride DMF (Drug Master File) is a document detailing the whole manufacturing process of Finasteride active pharmaceutical ingredient (API) in detail. Different forms of Finasteride DMFs exist exist since differing nations have different regulations, such as Finasteride USDMF, ASMF (EDMF), JDMF, CDMF, etc.

A Finasteride DMF submitted to regulatory agencies in the US is known as a USDMF. Finasteride USDMF includes data on Finasteride's chemical properties, information on the facilities and procedures used, and details about packaging and storage. The Finasteride USDMF is kept confidential to protect the manufacturer’s intellectual property.

click here to find a list of Finasteride suppliers with USDMF on PharmaCompass.

Finasteride JDMF

The Pharmaceuticals and Medical Devices Agency (PMDA) established the Japan Drug Master File (JDMF), also known as the Master File (MF), to permit Japanese and foreign manufacturers of drug substances, intermediates, excipients, raw materials, and packaging materials (‘Products’) to voluntarily register confidential information about the production and management of their products in Japan.

The Finasteride Drug Master File in Japan (Finasteride JDMF) empowers Finasteride API manufacturers to present comprehensive information (e.g., production methods, data, etc.) to the review authority, i.e., PMDA (Pharmaceuticals & Medical Devices Agency).

PMDA reviews the Finasteride JDMF during the approval evaluation for pharmaceutical products. At the time of Finasteride JDMF registration, PMDA checks if the format is accurate, if the necessary items have been included (application), and if data has been attached.

click here to find a list of Finasteride suppliers with JDMF on PharmaCompass.

Finasteride KDMF

In Korea, the Ministry of Food and Drug Safety (MFDS) is in charge of regulating pharmaceutical products and services.

Pharmaceutical companies submit a Finasteride Drug Master File in Korea (Finasteride KDMF) to the MFDS, which includes comprehensive information about the production, processing, facilities, materials, packaging, and testing of Finasteride. The MFDS reviews the Finasteride KDMF as part of the drug registration process and uses the information provided in the Finasteride KDMF to evaluate the safety and efficacy of the drug.

After submitting a Finasteride KDMF to the MFDS, the registered manufacturer can provide importers or distributors with the registration number without revealing confidential information to Korean business partners. Applicants seeking to register their Finasteride API can apply through the Korea Drug Master File (KDMF).

click here to find a list of Finasteride suppliers with KDMF on PharmaCompass.

Finasteride CEP

A Finasteride CEP of the European Pharmacopoeia monograph is often referred to as a Finasteride Certificate of Suitability (COS). The purpose of a Finasteride CEP is to show that the European Pharmacopoeia monograph adequately controls the purity of Finasteride EP produced by a given manufacturer. Suppliers of raw materials can prove the suitability of Finasteride to their clients by showing that a Finasteride CEP has been issued for it. The manufacturer submits a Finasteride CEP (COS) as part of the market authorization procedure, and it takes on the role of a Finasteride CEP holder for the record. Additionally, the data presented in the Finasteride CEP (COS) is managed confidentially and offers a centralized system acknowledged by numerous nations, exactly like the Finasteride DMF.

A Finasteride CEP (COS) is recognised by all 36 nations that make up the European Pharmacopoeia Convention. Finasteride CEPs may be accepted in nations that are not members of the Ph. Eur. at the discretion of the authorities there.

click here to find a list of Finasteride suppliers with CEP (COS) on PharmaCompass.

Finasteride WC

A Finasteride written confirmation (Finasteride WC) is an official document issued by a regulatory agency to a Finasteride manufacturer, verifying that the manufacturing facility of a Finasteride active pharmaceutical ingredient (API) adheres to the Good Manufacturing Practices (GMP) regulations of the importing country. When exporting Finasteride APIs or Finasteride finished pharmaceutical products to another nation, regulatory agencies frequently require a Finasteride WC (written confirmation) as part of the regulatory process.

click here to find a list of Finasteride suppliers with Written Confirmation (WC) on PharmaCompass.

Finasteride NDC

National Drug Code is a comprehensive database maintained by the FDA that contains information on all drugs marketed in the US. This directory includes information about finished drug products, unfinished drug products, and compounded drug products, including those containing Finasteride as an active pharmaceutical ingredient (API).

Finished drug products

The FDA updates the NDC directory daily. The NDC numbers for Finasteride API and other APIs are published in this directory by the FDA.

Unfinished drugs

The NDC unfinished drugs database includes product listing information submitted for all unfinished drugs, such as active pharmaceutical ingredients (APIs), drugs intended for further processing and bulk drug substances for compounding.

Pharmaceutical companies that manufacture Finasteride as an active pharmaceutical ingredient (API) must furnish the FDA with an updated record of all drugs that they produce, prepare, propagate, compound, or process for commercial distribution in the US at their facilities.

Compounded drug products

The NDC directory also contains data on finished compounded human drug products that contain Finasteride and are produced by outsourcing facilities. While these outsourcing facilities are not mandated to assign a Finasteride NDC to their finished compounded human drug products, they may choose to do so.

click here to find a list of Finasteride suppliers with NDC on PharmaCompass.

Finasteride GMP

Finasteride Active pharmaceutical ingredient (API) is produced in GMP-certified manufacturing facility.

GMP stands for Good Manufacturing Practices, which is a system used in the pharmaceutical industry to make sure that goods are regularly produced and monitored in accordance with quality standards. The FDA’s current Good Manufacturing Practices requirements are referred to as cGMP or current GMP which indicates that the company follows the most recent GMP specifications. The World Health Organization (WHO) has its own set of GMP guidelines, called the WHO GMP. Different countries can also set their own guidelines for GMP like China (Chinese GMP) or the EU (EU GMP).

PharmaCompass offers a list of Finasteride GMP manufacturers, exporters & distributors, which can be sorted by USDMF, JDMF, KDMF, CEP (COS), WC, API price, and more, enabling you to easily find the right Finasteride GMP manufacturer or Finasteride GMP API supplier for your needs.

Finasteride CoA

A Finasteride CoA (Certificate of Analysis) is a formal document that attests to Finasteride's compliance with Finasteride specifications and serves as a tool for batch-level quality control.

Finasteride CoA mostly includes findings from lab analyses of a specific batch. For each Finasteride CoA document that a company creates, the USFDA specifies specific requirements, such as supplier information, material identification, transportation data, evidence of conformity and signature data.

Finasteride may be tested according to a variety of international standards, such as European Pharmacopoeia (Finasteride EP), Finasteride JP (Japanese Pharmacopeia) and the US Pharmacopoeia (Finasteride USP).

Inform the supplier about your product requirements, specifying if you need a product with particular monograph like EP (Ph. Eur.), USP, JP, BP, or any other quality. In addition, clarify whether you need hydrochloride (HCl), anhydricum, base, micronisatum or a specific level of purity. To find reputable suppliers, utilize the filters and select those certified by GMP, FDA, or any other certification as per your requirement.
For your convenience, we have listed synonyms and CAS numbers to help you find the best supplier. The use of synonyms and CAS numbers can be helpful in identifying potential suppliers, but it is crucial to note that they might not always indicate the exact same product. It is important to confirm the product details with the supplier before making a purchase to ensure that it meets your requirements.

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ACTIVE PHARMA INGREDIENTS

30 API Suppliers

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32 KDMF

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$ API Reference Price

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135 Europe

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DRUG PRODUCT COMPOSITIONS

TABLET;ORAL - 5MG

TABLET;ORAL - 1MG

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