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Find Clinical Drug Pipeline Developments & Deals by BioArctic AB
Details :
Under the license agreement, BMS will become solely responsible for the development and any subsequent commercialization of BAN1503, which is being evaluated for Alzheimer Disease.
Details :
Leqembi (lecanemab) is an IgG1 monoclonal antibody directed against aggregated soluble and insoluble forms of amyloid-beta. It is being investigated for the treatment of early Alzheimer’s disease.
Details :
Leqembi (lecanemab) is an IgG1 monoclonal antibody directed against aggregated soluble (protofibril) and insoluble forms of amyloid-beta. It is indicated for the treatment of Alzheimer’s disease.
Details :
Leqembi (lecanemab) is an IgG1 monoclonal antibody directed against aggregated soluble (protofibril) and insoluble forms of amyloid-beta. It is indicated for the treatment of Alzheimer’s disease.
Details :
Leqembi (lecanemab) is an IgG1 monoclonal antibody directed against aggregated soluble (protofibril) and insoluble forms of amyloid-beta. It is indicated for the treatment of Alzheimer’s disease.
Details :
The agreement aims to evaluate BAN2802, a potential new treatment combining BioArctic's proprietary BrainTransporter™ technology with an undisclosed Alzheimer drug candidate.
Details :
Leqembi (lecanemab-irmb) is a humanized IgG1 monoclonal antibody targeting amyloid-beta, being evaluated for IV maintenance dosing in early Alzheimer's disease treatment.
Details :
Leqembi (lecanemab-irmb) is a humanized IgG1 monoclonal antibody directed against aggregated soluble and insoluble amyloid-beta forms. It is indicated for the treatment of Alzheimer’s disease.
Details :
Leqembi (lecanemab-irmb) is a humanized immunoglobulin gamma 1 (IgG1) monoclonal antibody directed against aggregated soluble (protofibril) and insoluble forms of amyloid-beta (Aβ). It is indicated for the treatment of Alzheimer’s disease.
Details :
Lecanemab is an investigational humanized monoclonal antibody for AD. Lecanemab selectively binds and eliminates soluble, toxic amyloid-beta (Aβ) aggregates (protofibrils) that are thought to contribute to the neurotoxicity in AD.
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